DIFFUSIVE ASEPTICAL PODDERMATIT, PURULENT SUBDERMATITIS, Superficial suppurative pododermatitis - Veterinary orthopedics


Diffuse aseptic pododermatitis (pododermatitis aseptica diffusa) develops as a result of mechanical influences on the entire ungulate sole or on all hooves. It is relatively rare.

Etiology. The compression and shaking of the basis of the hoof skin and its serous or serous-fibrinous inflammation is possible with prolonged distillation of animals over hard ground, their prolonged transport, especially under shaking conditions,

otelnoe overexposure of one limb in case of serious illness, another, with loose cattle on concrete floors with strong abrasive properties, when the hoof horn wears out faster than it grows.

Pathogenesis. As a result of strong mechanical action in the vascular layer, acute aseptic, serous or serous-fibrinous inflammation develops, spreading to all layers of the hoof skin (in depth), as well as to neighboring areas. Exudative phenomena develop. Exudate, accumulated between the horny layer of the epidermis and the hoof bone, compresses the nerve endings, causing severe soreness, and in the future the horn can exfoliate from the papillary (leaf) layer and move up and back, breaking outward in the corolla region. The microflora penetrates into the formed slits, which can lead to purulent pododermatitis and complete collapse of the horn shoe.

Clinical signs. If one or more of the extremities are involved, the animal lays more, supports the weight of the body to a healthy limb when supported. During movement, severe lameness of the resting type is manifested. The hoofs are hot, very painful, the pulsation of the digital arteries increases.

The prognosis for timely treatment is favorable, when the horny shoe is detached unfavorably.

Diagnosis. Diagnosis is based on clinical signs.

Treatment. In the first 24 ... 48 hours, cold is applied locally, further heat procedures (baths, clay, mud therapy, UHF) are recommended. Daily, intravenous injection of 0.25% solution of novocaine in a dose of 2 ml/kg body weight of the animal, 10% calcium chloride solution.


Superficial purulent pododermatitis

The superficial suppurative pododermatitis (pododermatitis purulenta superficialis) occurs, as a rule, as a result of damage to the basis of the hoof skin with violation of the integrity of the horn capsule and the subsequent penetration of the microflora into the skin.


Pathogenesis. The inflammatory process is localized in the surface layer of the skin's base and the producing layer of the epidermis, accompanied by a disorder of the circulation, a malnutrition, degenerative changes and purulent tissue melting.

Owing to the relatively small migration of leukocytes, the pus has a liquid consistency; it is painted in gray or dark gray color, depending on the admixture of horn pigments. With pododermatitis in the side wall area, the purulent exudate rises up between the horny leaves and the base skin sheets and can accumulate over the coronal edge of the horny capsule. Due to the destruction of leaflets and papillae, the horn flakes from the underlying tissues. When pododermatite soles pus accumulates in the white line; This is partly facilitated by the concavity of the sole.

Clinical signs. The local temperature of the hoof and the overall body temperature are increased. There is a pain when pressing with hoof forceps in the area of ​​the affected area. The limb of the resting limb develops relatively quickly, when a purulent exudate, accumulated between the surface of the skin base and the horn capsule, begins to exert pressure on the underlying tissues. Purulent exudate when exiting it outward spontaneously or at dissection is a liquid gray or dark gray color with an unpleasant smell (due to the decomposition of the horn, hydrogen sulphide is formed).

With superficial purulent pododermatitis, the following outcomes are possible:

resorption of exudate and regeneration of destroyed tissues (with mild forms of pododermatitis);

the formation of an abscess, usually in the area of ​​the corolla and crumbs, and its subsequent independent dissection due to tissue decay; often the abscess is opened with an operative pug;

spread of the inflammatory process to the underlying layers (deep pododermatitis); such an outcome is noted as a result of untimely and incorrect treatment.

The forecast is favorable in most cases. With the formation of an abscess in the area of ​​the corolla or crumbs, the course of the painful process is somewhat complicated. The transition of the inflammatory process from the surface to the deeper layers worsens the prognosis.

Diagnostics. The diagnosis is based on clinical signs.

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