Inflammation of the venous joint, Aseptic synovitis of the coronary artery, Purulent inflammation of the coronary artery, Deforming arthritis of the coronary artery - Veterinary orthopedics

INFLAMMATION OF THE VENOUS JOINT

Inflammation of the coronary arthritis (phalangis secundae) can occur as an aseptic synovitis, purulent arthritis and deforming and ossifying periarthritis of the coronary joint.

Aseptic synovitis of the coronary joint

Etiology. Aseptic synovitis of the coronary joint develops due to injury or stretching.

Pathogenesis and clinical signs. When acute flow observed mixed lameness. Note the swelling, which increases during the first 2 ... 3 days, very painful.

The chronic course of synovitis is characterized by the accumulation of fluid exudate in the joint cavity, an increase in its volume.

The accumulation of a significant amount of exudate is accompanied by increased fatigue, the animal often stumbles. Usually the observed symptoms (lameness of a significant degree with predominance of abnormalities of the support function, swelling at the joint level, soreness with a rotational sample) is not enough for an accurate diagnosis. The same signs occur when the tendon is stretched, the fracture of the coronary bone.

Diagnostics. A definite value in differential diagnosis is arthropupuncture and the preparation of aseptic exudate. Consider that his sweat may be a consequence of the extension of the joint.

Treatment. In each individual case, it is necessary to focus specifically on the extension of the joint, using appropriate means of its prevention and therapy.

Purulent inflammation of the coronary joint

Etiology. Purulent inflammation of the coronary artery (inflammation purulentae articulationis phalangis secundae) arises as a result of infected penetrating joint wounds, as well as in severe phlegmon of the corolla region, less often - metastatically as a result of all sorts of mechanical injuries.

Pathogenesis. Infection of the coronary joint is usually accompanied by the development of a capsular phlegmon. This is facilitated by the spread of the process in the deep tissues without the predisposition to spontaneous breakthrough, which is impeded by ligaments and tendons, tightly covering the joint in a circle, on top of them there is a wide palatine and plantar fascia and thickened skin in this section. Often the purulent process develops simultaneously in the common tendon vagina of the finger and the hoof joint.

Clinical signs. Purulent arthritis is severe. In a sick animal, the lameness of the resting limb of the third degree is noted, the animal jumps on three limbs. The general condition is depressed.

Diagnostics. The diagnosis is based on clinical signs.

Treatment. It should be noted that complications with purulent inflammation of the coronary artery occur much more often than with a similar joint disease. In most cases

The purulent inflammation of the coronary joint in the complication of the capsular, paraarticular flegmon and purulent osteoarthritis in horses is incurable.

Deforming arthritis of the coronary artery

Deforming arthritis of the coronary arthritis deformans phalangis secundae proceeds brightly.

Etiology. The venous bone of the spongy structure, small in volume, suffers considerably when the limb is overloaded, concussions on hard ground, when the turn is forced, etc. Especially severe consequences of these influences are noted when salt and vitamin metabolism is disturbed, especially in young animals. More often the disease occurs on the thoracic extremities.

Pathogenesis and clinical signs. At the beginning of the disease, there is lameness, which decreases in work. Later, during the second month of the disease, with the growth of the periosteum at the articular edge of the prostate and coronary artery, the diagnosis is made much easier by identifying the localized joint swelling in the joints, the symptoms of arthrogenic contracture (the fossa assumes a more perpendicular position), and the mobility of the coronary joint. A long-term consequence of the deforming inflammation of the coronary joint is true ankylosis. Structural changes during the process (periosteal growths, lacunar resorption, later osteosclerosis, changes in the joint gap) are determined by radiography.

The prognosis is cautious, since the animal is able to move by step, which is taken into account while preserving valuable in the pedigree relation of females and males.

Diagnostics. The diagnosis is based on clinical signs. In order to differentiate from chronic bezexudative processes in other joints, a novocaine blockade is used.

Treatment. Similar to that with ossifying periarthritis, chronic sesamoiditis, deforming inflammation of the prosthetic joint.

2.16.2.4. Ossifying periarthritis of the coronary artery

Ossifying periarthritis of the coronary joint (periarthritis ossificans phalangis secundae) is often observed in horses.

Etiology. To the causes of ossifying periarthritis relative

Siyat open and closed lesions of periarticular tissues and periosteum, for example, with wounds and bruises; fractures and cracks of the coronary and putt bones; stretching and tearing of the ligaments of the joint; phlegmon in the fetal area; tendinitis and tendovaginitis of the distal limbs; periarticular fibrositis, rheumatic arthritis and osteodystrophic lesions of bone tissue.

Other lesions with ruptures of the joint capsule and ligaments (dislocation, fracture of the coronary bone) are also accompanied by periarthritis. Less often it accompanies purulent processes, such as pararticular, capsular phlegmon, purulent periostitis.

Pathogenesis. There is a proliferation of periosteum of the joint margin and ossification of the proliferative, more pronounced from the coronoid bone. Dimensions of hyperostosis of the distal pituitary epiphysis are usually more limited.

Clinical signs. With ossifying arthritis painful process develops gradually. In the case of acute course of the disease, palpation in the region of the coronary joint reveals a moderate, dense, elastic swelling. Passive movements are painful. In a calm state, the animal holds the aching limb of the finger bent in the joints and gently rests on the soil with the hoof wall. When moving, the lameness of the resting limb arises, which at the beginning of the work appears stronger, then gradually decreases, and sometimes even completely stops.

In the chronic course of the process, periarticular changes in the joint are more pronounced: the joint is thickened, deformed; Its swelling is firm and almost painless; passive movements are limited. Lame is slightly expressed. When palpation, exostoses are found. The mobility of the joint is limited, which is associated with stratification of the proliferative periosteum, para-articular fibrositis, ossification of ligaments and tendons. Progression of the process is completed with ankylosis.

The forecast is unfavorable, since functional violations are unrecoverable. The horse can be used in the work step.

Diagnostics. Diagnosis is based on clinical signs and is refined by radiography.

Treatment. With periostitis and ossifying arthritis of the coronary joint, the animal is released from work. In cases of acute fibrous periostitis, warming compresses, iontophoresis of iodine, moxibustion are administered to the affected area during the first 3 days. In chronic ossifying periostitis, rubbing of irritating ointments, moxibustion is shown.

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